human hdl (Lee Biosolutions)
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Human Hdl, supplied by Lee Biosolutions, used in various techniques. Bioz Stars score: 92/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+hdl/Human+Plasma+High+Density+Lipoprotein+-+Sterile+Filtered/pmc12627944-141-16-18
Average 92 stars, based on 6 article reviews
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Concentration Assay:Article Title: Angiopoietin-like 3 inhibition of endothelial lipase is not modulated by angiopoietin-like 8 Article Snippet: .. EL (final concentration of ∼60 μg/ml) and ANGPTL proteins (0–2,600 ng/ml ANGPTL3; 0–78 ng/ml ANGPTL8) were incubated together at 37°C for 30 min. Article Title: Compromised lipid metabolism, mitochondria respiration and neuroprotective effects in iPSC-derived astrocytes from a Smith-Lemli-Opitz syndrome patient Article Snippet: NPCs were either cryopreserved in StemMACS cryo-brew (Miltenyi Biotec, 130–109-558) or expanded in neural precursor media containing DMEM, B-27 without vitamin A, bFGF (20 ng/ml), EGF (20 ng/ml), glutamine (2 mM), and penicillin–streptomycin (50 μg/ml). .. To allow SLOS NPC formation and proliferation [ ], sterile-filtered human LDL (Lee Biosolutions, 360–12) and Article Title: Compromised lipid metabolism, mitochondria respiration and neuroprotective effects in iPSC-derived astrocytes from a Smith-Lemli-Opitz syndrome patient. Article Snippet: Smith-Lemli-Opitz syndrome (SLOS) is a rare, autosomal recessive disorder characterized by congenital malformations, intellectual disability, and behavioral abnormalities.. SLOS results from mutations in the DHCR7 gene, leading to impaired cholesterol biosynthesis due to dysregulation of 7-dehydrocholesterol reductase.. Cholesterol plays crucial roles in neurophysiology, including synaptic formation and neurotransmitter receptor regulation, which likely contribute to neurological manifestations in SLOS patients. Incubation:Article Title: Angiopoietin-like 3 inhibition of endothelial lipase is not modulated by angiopoietin-like 8 Article Snippet: .. EL (final concentration of ∼60 μg/ml) and ANGPTL proteins (0–2,600 ng/ml ANGPTL3; 0–78 ng/ml ANGPTL8) were incubated together at 37°C for 30 min. Article Title: The Regulation of COX-2, ABCA1 and ABCG1 by the lncRNA PACERR links the inflammatory response and cholesterol homeostasis Article Snippet: .. Cells were labeled with 0.5 μCi ml −1 of H-cholesterol (PerkinElmer) and 37.5 μg ml −1 acetylated LDL (Kalen Biomedical, LLC 770201-6) for 24 h, washed twice with PBS and then incubated with 2 mg ml −1 fatty-acid-free BSA (Sigma-Aldrich) in medium for 24 h. To induce efflux, we added 50 μg ml −1 Article Title: Endothelial Lipase Variant, T111I, Does Not Alter Inhibition by Angiopoietin-like Proteins Article Snippet: .. EL conditioned media and 67 or 116.4 mg/dL (cholesterol) of Labeling:Article Title: The Regulation of COX-2, ABCA1 and ABCG1 by the lncRNA PACERR links the inflammatory response and cholesterol homeostasis Article Snippet: .. Cells were labeled with 0.5 μCi ml −1 of H-cholesterol (PerkinElmer) and 37.5 μg ml −1 acetylated LDL (Kalen Biomedical, LLC 770201-6) for 24 h, washed twice with PBS and then incubated with 2 mg ml −1 fatty-acid-free BSA (Sigma-Aldrich) in medium for 24 h. To induce efflux, we added 50 μg ml −1 Sterility:Article Title: Compromised lipid metabolism, mitochondria respiration and neuroprotective effects in iPSC-derived astrocytes from a Smith-Lemli-Opitz syndrome patient Article Snippet: NPCs were either cryopreserved in StemMACS cryo-brew (Miltenyi Biotec, 130–109-558) or expanded in neural precursor media containing DMEM, B-27 without vitamin A, bFGF (20 ng/ml), EGF (20 ng/ml), glutamine (2 mM), and penicillin–streptomycin (50 μg/ml). .. To allow SLOS NPC formation and proliferation [ ], sterile-filtered human LDL (Lee Biosolutions, 360–12) and Article Title: Compromised lipid metabolism, mitochondria respiration and neuroprotective effects in iPSC-derived astrocytes from a Smith-Lemli-Opitz syndrome patient. Article Snippet: Smith-Lemli-Opitz syndrome (SLOS) is a rare, autosomal recessive disorder characterized by congenital malformations, intellectual disability, and behavioral abnormalities.. SLOS results from mutations in the DHCR7 gene, leading to impaired cholesterol biosynthesis due to dysregulation of 7-dehydrocholesterol reductase.. Cholesterol plays crucial roles in neurophysiology, including synaptic formation and neurotransmitter receptor regulation, which likely contribute to neurological manifestations in SLOS patients. |
